High Glucose-Induced Thioredoxin-Interacting Protein in Renal Proximal Tubule Cells Is Independent of Transforming Growth Factor-β1

This publication has 42 references indexed in Scilit:

• Therapeutic Approach for Diabetic Nephropathy Using Gene Delivery of Translocase of Inner Mitochondrial Membrane 44 by Reducing Mitochondrial Superoxide Production
  Journal of the American Society of Nephrology, 2006
• Kidney involvement in a nongenetic rat model of type 2 diabetes
  Kidney International, 2005
• Connective tissue growth factor mediates the profibrotic effects of transforming growth factor-β produced by tubular epithelial cells in response to high glucose
  Clinical and Experimental Nephrology, 2005
• Plasminogen activator inhibitor-1 deficiency retards diabetic nephropathy
  Kidney International, 2005
• Transforming Growth Factor β–SMAD2 Signaling Regulates Aortic Arch Innervation and Development
  Circulation Research, 2004
• The effect of high glucose and PPAR-γ agonists on PPAR-γ expression and function in HK-2 cells
  American Journal of Physiology-Renal Physiology, 2004
• TGF‐β3–dependent SMAD2 phosphorylation and inhibition of MEE proliferation during palatal fusion
  Developmental Dynamics, 2003
• A new mathematical model for relative quantification in real-time RT-PCR
  Nucleic Acids Research, 2001
• Effects of endothelin or angiotensin II receptor blockade on diabetes in the transgenic (mRen-2)27 rat
  Kidney International, 2000
• Matrilysin: An epithelial matrix metalloproteinase with potentially novel functions
  The International Journal of Biochemistry & Cell Biology, 1996