Role of Smad proteins in the regulation of NF-κB by TGF-β in colon cancer cells
- 31 July 2006
- journal article
- Published by Elsevier in Cellular Signalling
- Vol. 18 (7) , 1041-1050
- https://doi.org/10.1016/j.cellsig.2005.08.021
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- The function of multiple IκB : NF-κB complexes in the resistance of cancer cells to Taxol-induced apoptosisOncogene, 2002
- Transforming Growth Factor-β Repression of Matrix Metalloproteinase-1 in Dermal Fibroblasts Involves Smad3Journal of Biological Chemistry, 2001
- Transforming Growth Factor-β1 Promotes Invasiveness after Cellular Transformation with Activated Ras in Intestinal Epithelial CellsExperimental Cell Research, 2001
- Smad7 inhibits the survival nuclear factor κB and potentiates apoptosis in epithelial cellsOncogene, 2001
- Interaction and Functional Cooperation of NF-κB with SmadsJournal of Biological Chemistry, 2000
- Smad4/DPC4 Silencing and Hyperactive Ras Jointly Disrupt Transforming Growth Factor-β Antiproliferative Responses in Colon Cancer CellsJournal of Biological Chemistry, 1999
- Overexpression of urokinase-type plasminogen activator in pancreatic adenocarcinoma is regulated by constitutively activated RelAOncogene, 1999
- Transformation of intestinal epithelial cells by chronic TGF-β1 treatment results in downregulation of the type II TGF-β receptor and induction of cyclooxygenase-2Oncogene, 1999
- TGFβ signaling is necessary for carcinoma cell invasiveness and metastasisCurrent Biology, 1998
- Signaling Activity of Homologous and Heterologous Transforming Growth Factor-β Receptor Kinase ComplexesJournal of Biological Chemistry, 1995