D1-receptor-related priming is attenuated by antisense-meditated `knockdown' of fosB expression
- 1 January 1998
- journal article
- Published by Elsevier in Molecular Brain Research
- Vol. 53 (1-2) , 69-77
- https://doi.org/10.1016/s0169-328x(97)00281-7
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Chronic Alterations in Dopaminergic Neurotransmission Produce a Persistent Elevation of ΔFosB‐like Protein(s) in both the Rodent and Primate StriatumEuropean Journal of Neuroscience, 1996
- Striatal c‐fos Induction by Cocaine or Apomorphine Occurs Preferentially in Output Neurons Projecting to the Substantia Nigra in the RatEuropean Journal of Neuroscience, 1992
- Both products of the fosB gene, FosB and its short form, FosB/SF, are transcriptional activators in fibroblasts.Molecular and Cellular Biology, 1991
- Expression of different Jun and Fos proteins during the G0-to-G1 transition in mouse fibroblasts: in vitro and in vivo associations.Molecular and Cellular Biology, 1991
- Amphetamine and cocaine induce drug-specific activation of the c-fos gene in striosome-matrix compartments and limbic subdivisions of the striatum.Proceedings of the National Academy of Sciences, 1990
- fra-1: a serum-inducible, cellular immediate-early gene that encodes a fos-related antigen.Molecular and Cellular Biology, 1988
- Agonist-induced homologous and heterologous sensitization to D-1- and D-2-dependent contraversive turningEuropean Journal of Pharmacology, 1987
- Behavioural expression of D-1 receptor supersensitivity depends on previous stimulation of D-2 receptorsLife Sciences, 1987
- Sensory inattention produced by 6-hydroxydopamine-induced degeneration of ascending dopamine neurons in the brainExperimental Neurology, 1976
- Nigrostriatal bundle damage and the lateral hypothalamic syndrome.Journal of Comparative and Physiological Psychology, 1974