Activated Ras induces cytoplasmic vacuolation and non-apoptotic death in glioblastoma cells via novel effector pathways
- 31 May 2007
- journal article
- Published by Elsevier in Cellular Signalling
- Vol. 19 (5) , 1034-1043
- https://doi.org/10.1016/j.cellsig.2006.11.010
Abstract
No abstract availableKeywords
This publication has 75 references indexed in Scilit:
- Gene silencing reveals a specific function of hVps34 phosphatidylinositol 3-kinase in late versus early endosomesJournal of Cell Science, 2006
- Cell Type-Specific Tumor Suppression by Ink4a and Arf in Kras-Induced Mouse GliomagenesisCancer Research, 2005
- Activation of H-Ras in the Endoplasmic Reticulum by the RasGRF Family Guanine Nucleotide Exchange FactorsMolecular and Cellular Biology, 2004
- The Pro-apoptotic Ras Effector Nore1 May Serve as a Ras-regulated Tumor Suppressor in the LungJournal of Biological Chemistry, 2003
- Oncogenic Ras triggers cell suicide through the activation of a caspase-independent cell death program in human cancer cellsOncogene, 1999
- Evidence for a Dual Control of Macroautophagic Sequestration and Intracellular Trafficking of N-Linked Glycoproteins by the Trimeric Gi3Protein in HT-29 CellsBiochemical and Biophysical Research Communications, 1997
- Stimulation of Membrane Ruffling and MAP Kinase Activation by Distinct Effectors of RASScience, 1996
- Identification of AF-6 and Canoe as Putative Targets for RasJournal of Biological Chemistry, 1996
- Phosphatidylinositol-3-OH kinase direct target of RasNature, 1994
- Biological and biochemical properties of human rasH genes mutated at codon 61Cell, 1986