Dexamethasone Induces Posttranslational Degradation of GLUT2 and Inhibition of Insulin Secretion in Isolated Pancreatic β Cells

This publication has 41 references indexed in Scilit:

• Defective Fatty Acid-mediated β-Cell Compensation in Zucker Diabetic Fatty Rats
  Journal of Biological Chemistry, 1996
• Long-term exposure of rat pancreatic islets to fatty acids inhibits glucose-induced insulin secretion and biosynthesis through a glucose fatty acid cycle.
  Journal of Clinical Investigation, 1994
• GLUT2 surface expression and intracellular transport via the constitutive pathway in pancreatic beta cells and insulinoma: evidence for a block in trans-Golgi network exit by brefeldin A.
  The Journal of cell biology, 1993
• GLUT-2 function in glucose-unresponsive beta cells of dexamethasone-induced diabetes in rats.
  Journal of Clinical Investigation, 1993
• What if Minkowski Had Been Ageusic? An Alternative Angle on Diabetes
  Science, 1992
• Mechanisms decreasing glucose oxidation in diabetes and starvation: Role of lipid fuels and hormones
  Diabetes/Metabolism Research and Reviews, 1988
• Cloning and functional expression in bacteria of a novel glucose transporter present in liver, intestine, kidney, and β-pancreatic islet cells
  Cell, 1988
• REPRODUCIBLE HIGH YIELD OF RAT ISLETS BY STATIONARY IN VITRO DIGESTION FOLLOWING PANCREATIC DUCTAL OR PORTAL VENOUS COLLAGENASE INJECTION1
  Transplantation, 1987
• Immunochemical evidence for a transmembrane orientation of both the (sodium(1+), potassium(1+) ion-activated)-ATPase subunits
  Biochemistry, 1981
• The Glucose Receptor A DEFECTIVE MECHANISM IN DIABETES MELLITUS DISTINCT FROM THE BETA ADRENERGIC RECEPTOR
  Journal of Clinical Investigation, 1973