• 1 January 1983
    • journal article
    • research article
    • Vol. 44  (8) , 1512-1515
Abstract
Gastric dilatation-volvulus (at 30 mm of Hg of gastric pressure) was experimentally induced in 8 dogs under sodium pentobarbital anesthesia. Hemodynamic indices including cardiac output, mean aortic pressure, left ventricular end diastolic pressure, heart rate, total peripheral resistance, and dp/dtmax [maximum value of ventricular pressure change] were measured during a 180-min period of gastric dilatation-volvulvus and for 120 min after gastric decompression. Experimental gastric dilatation-volvulus resulted in significant (P < 0.05) decreases in cardiac output (64%), mean aortic pressure (48%), left ventricular end diastolic pressure (68%), and ventricular dp/dtmax (32%) compared to 4 control dogs. Hemodynamic indices returned toward control values after gastric decompression. Gastric dilatation-volvulus in the presence experiment was capable of inducing hemodynamic alterations of a magnitude similar to those reported in gastric dilatation studies, using higher gastric pressures.

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