β-Adrenergic agonist-induced long-lasting synaptic modifications in hippocampal dentate gyrus require activation of NMDA receptors, but not electrical activation of afferents
- 1 September 1990
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 526 (2) , 347-350
- https://doi.org/10.1016/0006-8993(90)91245-c
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- NMDA receptor antagonists reduce medial, but not lateral, perforant path-evoked EPSPs in dentate gyrus of rat hippocampal sliceExperimental Brain Research, 1990
- Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiationNature, 1989
- NMDA receptor antagonists block norepinephrine-induced long-lasting potentiation and long-term potentiation in rat dentate gyrusBrain Research, 1989
- Noradrenaline and β-adrenoceptor agonists increase activity of voltage-dependent calcium channels in hippocampal neuronsNature, 1987
- Long-term potentiation in the hippocampus using depolarizing current pulses as the conditioning stimulus to single volley synaptic potentialsJournal of Neuroscience, 1987
- Long-term potentiation in the dentate gyrus: induction and increased glutamate release are blocked by d(−)aminophosphonovalerateNeuroscience, 1987
- The action of norepinephrine in the dentate gyrus: Beta-mediated facilitation of evoked potentials in vitroBrain Research, 1985
- Long-term potentiation in the hippocampus involves activation of N-methyl-D-aspartate receptorsBrain Research, 1984
- Inhibitory modulation of long-term potentiation: Evidence for a postsynaptic locus of controlBrain Research, 1982
- Long‐lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant pathThe Journal of Physiology, 1973