RalGDS is required for tumor formation in a model of skin carcinogenesis
- 1 March 2005
- journal article
- research article
- Published by Elsevier in Cancer Cell
- Vol. 7 (3) , 219-226
- https://doi.org/10.1016/j.ccr.2005.01.029
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Renewing the conspiracy theory debate: does Raf function alone to mediate Ras oncogenesis?Trends in Cell Biology, 2004
- Distinct requirements for Ras oncogenesis in human versus mouse cellsGenes & Development, 2002
- Mice deficient in the Rac activator Tiam1 are resistant to Ras-induced skin tumoursNature, 2002
- Mutations of the BRAF gene in human cancerNature, 2002
- The Activation of RalGDS Can Be Achieved Independently of Its Ras Binding DomainPublished by Elsevier ,2002
- The Brain Exocyst Complex Interacts with RalA in a GTP-dependent MannerPublished by Elsevier ,2001
- Ral GTPases Contribute to Regulation of Cyclin D1 through Activation of NF-κBMolecular and Cellular Biology, 2000
- RalGEF2, a Pleckstrin Homology Domain Containing Guanine Nucleotide Exchange Factor for RalPublished by Elsevier ,2000
- rsc: a novel oncogene with structural and functional homology with the gene family of exchange factors for RalOncogene, 1997
- Ra1 and Rab3a are major GTP-binding proteins of axonal rapid transport and synaptic vesicles and do not redistribute following depolarization stimulated synaptosomal exocytosisBiochimica et Biophysica Acta (BBA) - Biomembranes, 1993