Selective inactivation of redox-sensitive mitochondrial enzymes during cardiac reperfusion
- 20 September 2002
- journal article
- Published by Elsevier in Archives of Biochemistry and Biophysics
- Vol. 406 (2) , 222-228
- https://doi.org/10.1016/s0003-9861(02)00446-0
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Effects of myocardial ischemia and reperfusion on mitochondrial function and susceptibility to oxidative stressCellular and Molecular Life Sciences, 2001
- Declines in mitochondrial respiration during cardiac reperfusion: Age-dependent inactivation of α-ketoglutarate dehydrogenaseProceedings of the National Academy of Sciences, 1999
- Cardiac reperfusion injury: Aging, lipid peroxidation, and mitochondrial dysfunctionProceedings of the National Academy of Sciences, 1998
- Interrelationship between cellular calcium homeostasis and free radical generation in myocardial reperfusion injuryChemico-Biological Interactions, 1997
- Global ischaemia induces a biphasic response of the mitochondrial respiratory chain. Anoxic pre-perfusion protects against ischaemic damageBiochemical Journal, 1992
- The relationship between oxygen radical generation and impairment of myocardial energy metabolism following post-ischemic reperfusion,Journal of Molecular and Cellular Cardiology, 1991
- Reoxygenation-dependent decrease in mitochondrial NADH:CoQ reductase (Complex I) activity in the hypoxic/reoxygenated rat heartBiochemical Journal, 1991
- Direct measurement of free radical generation following reperfusion of ischemic myocardium.Proceedings of the National Academy of Sciences, 1987
- Development of ischemia-induced damage in defined mitochondrial subpopulationsJournal of Molecular and Cellular Cardiology, 1985
- Experimental myocardial ischemia characteristics of isolated mitochondrial subpopulationsJournal of Molecular and Cellular Cardiology, 1978