The Kindler Syndrome Protein Is Regulated by Transforming Growth Factor-β and Involved in Integrin-mediated Adhesion
Open Access
- 1 February 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (8) , 6824-6833
- https://doi.org/10.1074/jbc.m307978200
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- Smad-dependent and Smad-independent pathways in TGF-β family signallingNature, 2003
- Loss of Kindlin-1, a Human Homolog of the Caenorhabditis elegans Actin–Extracellular-Matrix Linker Protein UNC-112, Causes Kindler SyndromeAmerican Journal of Human Genetics, 2003
- Differential expression of transforming growth factors-β1, -β2 and -β3 in human colon carcinomaEuropean Journal Of Cancer, 2001
- The
UNC-112
Gene in Caenorhabditis elegansEncodes a Novel Component of Cell–Matrix Adhesion Structures Required for Integrin Localization in the Muscle Cell MembraneThe Journal of cell biology, 2000
- Transforming growth factor-β1 regulates basement membrane formation by alveolar epithelial cells in vitroEuropean Journal of Cell Biology, 1999
- TGFβ signaling is necessary for carcinoma cell invasiveness and metastasisCurrent Biology, 1998
- SWISS‐MODEL and the Swiss‐Pdb Viewer: An environment for comparative protein modelingElectrophoresis, 1997
- Absence of integrin α6 leads to epidermolysis bullosa and neonatal death in miceNature Genetics, 1996
- Integrin β4 mutations associated with junctional epidermolysis bullosa with pyloric atresiaNature Genetics, 1995
- Identification of a new protein localized at sites of cell-substrate adhesion.The Journal of cell biology, 1986