Multiple roles for PrP in the prion diseases

This publication has 9 references indexed in Scilit:

• Altered circadian activity rhythms and sleep in mice devoid of prion protein
  Nature, 1996
• Loss of cerebellar Purkinje cells in aged mice homozygous for a disrupted PrP gene
  Nature, 1996
• Role of microglia and host prion protein in neurotoxicity of a prion protein fragment
  Nature, 1996
• Normal host prion protein necessary for scrapie-induced neurotoxicity
  Nature, 1996
• GABAA, GABAC, and NMDA receptor subunit expression in the suprachiasmatic nucleus and other brain regions
  Molecular Brain Research, 1995
• Prion protein is necessary for normal synaptic function
  Nature, 1994
• 129/Ola mice carrying a null mutation in PrP that abolishes mRNA production are developmentally normal
  Molecular Neurobiology, 1994
• Mice devoid of PrP are resistant to scrapie
  Cell, 1993
• GABA regulation of circadian responses to light. I. Involvement of GABAA-benzodiazepine and GABAB receptors
  Journal of Neuroscience, 1989