Immune Activation of NF-κB and JNK Requires Drosophila TAK1
Open Access
- 1 December 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (49) , 48928-48934
- https://doi.org/10.1074/jbc.m304802200
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- TAK1 is Critical for IκB Kinase-mediated Activation of the NF-κB PathwayJournal of Molecular Biology, 2003
- Requirement for a Peptidoglycan Recognition Protein (PGRP) in Relish Activation and Antibacterial Immune Responses in DrosophilaScience, 2002
- Functional genomic analysis of phagocytosis and identification of a Drosophila receptor for E. coliNature, 2002
- The Drosophila immune response against Gram-negative bacteria is mediated by a peptidoglycan recognition proteinNature, 2002
- How Drosophila combats microbial infection: a model to study innate immunity and host–pathogen interactionsCurrent Opinion in Microbiology, 2002
- Activation of the JNK pathway during dorsal closure in Drosophila requires the mixed lineage kinase, slipperGenes & Development, 2002
- Regulation of Wnt Signaling and Embryo Patterning by an Extracellular SulfataseScience, 2001
- A Drosophila IκB kinase complex required for Relish cleavage and antibacterial immunityGenes & Development, 2000
- Phylogenetic Perspectives in Innate ImmunityScience, 1999
- In vitro induction of cecropin genes — an immune response in a Drosophila blood cell lineBiochemical and Biophysical Research Communications, 1992