Hypoglycaemia is a cause of axonal injury
- 1 October 2000
- journal article
- research article
- Published by Wiley in Neuropathology and Applied Neurobiology
- Vol. 26 (5) , 448-453
- https://doi.org/10.1046/j.1365-2990.2000.00273.x
Abstract
Axonal injury as demonstrated immunohistochemically is increasingly being recognized at post‐mortem in patients who have been unconscious, and in some cases the cause of the coma may not be immediate...Keywords
This publication has 19 references indexed in Scilit:
- The neuropathology of the vegetative state after head injuryJournal of Clinical Pathology, 1999
- Diffuse Axonal Injury: An Important Form of Traumatic Brain DamageThe Neuroscientist, 1998
- What's new in the diagnosis of head injury?Journal of Clinical Pathology, 1997
- Increased numbers of βAPP-immunoreactive neurones in the entorhinal cortex after head injuryNeuroReport, 1994
- Stalning af amyloid percursor protein to study axonal damage in mild head InjuryThe Lancet, 1994
- Beta amyloid protein deposition in the brain after severe head injury: implications for the pathogenesis of Alzheimer's disease.Journal of Neurology, Neurosurgery & Psychiatry, 1994
- β-Amyloid precursor protein (βAPP) as a marker for axonal injury after head injuryNeuroscience Letters, 1993
- Biological differences between ischemia, hypoglycemia, and epilepsyAnnals of Neurology, 1988
- BRAIN DAMAGE IN NON‐MISSILE HEAD INJURY SECONDARY TO HIGH INTRACRANIAL PRESSURE*Neuropathology and Applied Neurobiology, 1987
- THE RELATIONSHIP BETWEEN VENTRICULAR FLUID PRESSURE AND THE NEUROPATHOLOGY OF RAISED INTRACRANIAL PRESSURENeuropathology and Applied Neurobiology, 1976