The α3 and β4 nicotinic acetylcholine receptor subunits are necessary for nicotine-induced seizures and hypolocomotion in mice
- 30 September 2004
- journal article
- research article
- Published by Elsevier in Neuropharmacology
- Vol. 47 (3) , 401-407
- https://doi.org/10.1016/j.neuropharm.2004.05.002
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Increased Sensitivity to Agonist-Induced Seizures, Straub Tail, and Hippocampal Theta Rhythm in Knock-In Mice Carrying Hypersensitive α4 Nicotinic ReceptorsJournal of Neuroscience, 2003
- Increased Sensitivity to Nicotine-Induced Seizures in Mice Expressing the L250T α7 Nicotinic Acetylcholine Receptor MutationMolecular Pharmacology, 2002
- Retinal Neuron Activity of ETS Domain-binding Sites in a Nicotinic Acetylcholine Receptor Gene Cluster EnhancerJournal of Biological Chemistry, 2002
- Absence of α7-containing neuronal nicotinic acetylcholine receptors does not prevent nicotine-induced seizuresMolecular Brain Research, 2002
- Cellular mechanisms of nicotine addictionPharmacology Biochemistry and Behavior, 2001
- α10: A determinant of nicotinic cholinergic receptor function in mammalian vestibular and cochlear mechanosensory hair cellsProceedings of the National Academy of Sciences, 2001
- Nicotinic acetylcholine receptors in the autonomic control of bladder functionEuropean Journal of Pharmacology, 2000
- Neurobiological mechanisms by which nicotine mediates different types of anxietyEuropean Journal of Pharmacology, 2000
- The interpeduncular nucleus regulates nicotineʼs effects on free-field activityNeuroReport, 1995
- Nicotine: Effect on the Sleep Cycle of the CatScience, 1965