Protease inhibitors restore radiation-induced apoptosis to Bcl-2-expressing lymphoma cells.
- 19 October 2001
- journal article
- research article
- Published by Wiley in International Journal of Cancer
- Vol. 96 (6) , 327-333
- https://doi.org/10.1002/ijc.1040
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Enhancement of radiosensitivity by proteasome inhibition: Implications for a role of NF-κBInternational Journal of Radiation Oncology*Biology*Physics, 2001
- Roles of NF-κB and 26 S Proteasome in Apoptotic Cell Death Induced by Topoisomerase I and II Poisons in Human Nonsmall Cell Lung CarcinomaPublished by Elsevier ,2001
- Tumor Necrosis Factor-α-inducible IκBα Proteolysis Mediated by Cytosolic m-CalpainJournal of Biological Chemistry, 1999
- Prostate carcinoma cell death resulting from inhibition of proteasome activity is independent of functional Bcl-2 and p53Oncogene, 1998
- Novel dipeptidyl proteasome inhibitors overcome Bcl-2 protective function and selectively accumulate the cyclin-dependent kinase inhibitor p27 and induce apoptosis in transformed, but not normal, human fibroblastsCell Death & Differentiation, 1998
- Evidence against a functional site for Bcl-2 downstream of caspase cascade in preventing apoptosisOncogene, 1997
- Resistance to radiation-induced apoptosis in Bcl-2-expressing cells is reversed by depleting cellular thiolsOncogene, 1997
- THE NF-κB AND IκB PROTEINS: New Discoveries and InsightsAnnual Review of Immunology, 1996
- Is calpain activity regulated by membranes and autolysis or by calcium and calpastatin?BioEssays, 1992
- Degradation of transcription factors, c‐Jun and c‐Fos, by calpainFEBS Letters, 1991