Fas-Mediated Apoptosis in Cholangiocarcinoma Cells Is Enhanced by 3,3′-Diindolylmethane through Inhibition of AKT Signaling and FLICE-Like Inhibitory Protein

This publication has 48 references indexed in Scilit:

• Enhancement of susceptibility to Fas-mediated apoptosis in HL-60 cells through down-regulation of cellular FLICE-inhibitory protein by phosphatidylinositol 3-kinase inhibitors
  Oncology Reports, 2005
• Agonists of an ecdysone-inducible mammalian expression system inhibit Fas Ligand- and TRAIL-induced apoptosis in the human colon carcinoma cell line RKO
  Cell Death & Differentiation, 2005
• A Novel Ring-Substituted Diindolylmethane,1,1-Bis[3′-(5-Methoxyindolyl)]-1-(p-t-Butylphenyl) Methane, Inhibits Extracellular Signal-Regulated Kinase Activation and Induces Apoptosis in Acute Myelogenous Leukemia
  Cancer Research, 2005
• Predominant Bcl-XL Knockdown Disables Antiapoptotic Mechanisms: Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand–Based Triple Chemotherapy Overcomes Chemoresistance in Pancreatic Cancer Cells In vitro
  Cancer Research, 2005
• Cholangiocarcinoma
  Clinics in Liver Disease, 2001
• The caspase-8 inhibitor FLIP promotes activation of NF-κB and Erk signaling pathways
  Current Biology, 2000
• PI3-K/AKT Regulation of NF-κB Signaling Events in Suppression of TNF-Induced Apoptosis
  Biochemical and Biophysical Research Communications, 2000
• Apoptosis induced in normal human hepatocytes by tumor necrosis factor-related apoptosis-inducing ligand
  Nature Medicine, 2000
• Akt Suppresses Apoptosis by Stimulating the Transactivation Potential of the RelA/p65 Subunit of NF-κB
  Molecular and Cellular Biology, 2000
• Cholangiocarcinomas express fas ligand and disable the fas receptor
  Hepatology, 1999