Excretion of prostacyclin and thromboxane metabolites before, during, and after pregnancy-induced hypertension

This publication has 16 references indexed in Scilit:

• Endothelial vasoactive mediators in preeclampsia
  American Journal of Obstetrics and Gynecology, 1993
• Prostacyclin rather than nitric oxide lowers human umbilical artery tone in vitro
  European Journal of Obstetrics & Gynecology and Reproductive Biology, 1992
• Platelet activation and vascular damage in gestational hypertension
  American Journal of Obstetrics and Gynecology, 1992
• Thromboxane A2 synthesis in pregnancy-induced hypertension
  The Lancet, 1990
• Preeclampsia: An endothelial cell disorder
  American Journal of Obstetrics and Gynecology, 1989
• Blood-vessel wall arachidonate metabolism and its pharmacological modification in a new in vitro assay system
  Naunyn-Schmiedebergs Archiv für experimentelle Pathologie und Pharmakologie, 1988
• Increased thromboxane biosynthesis in normal pregnancy is mainly derived from platelets
  American Journal of Obstetrics and Gynecology, 1987
• Decreased prostacyclin biosynthesis preceding the clinical manifestation of pregnancy-induced hypertension.
  Circulation, 1987
• Renal Prostacyclin and Thromboxane in Normotensive and Preeclamptic Pregnant Women and Their Infants*
  Journal of Clinical Endocrinology & Metabolism, 1986
• Prostacyclin production during pregnancy: Comparison of production during normal pregnancy and pregnancy complicated by hypertension
  American Journal of Obstetrics and Gynecology, 1982