Flow cytometric measurement of phosphorylated STAT5 in AML: lack of specific association with FLT3 internal tandem duplications

This publication has 12 references indexed in Scilit:

• Constitutive activation of STAT3 and STAT5 is induced by leukemic fusion proteins with protein tyrosine kinase activity and is sufficient for transformation of hematopoietic precursor cells
  Experimental Hematology, 2002
• Regulation of constitutive STAT5 phosphorylation in acute myeloid leukemia blasts
  Leukemia, 2001
• The presence of a FLT3 internal tandem duplication in patients with acute myeloid leukemia (AML) adds important prognostic information to cytogenetic risk group and response to the first cycle of chemotherapy: analysis of 854 patients from the United Kingdom Medical Research Council AML 10 and 12 trials
  Blood, 2001
• Flt3 mutations from patients with acute myeloid leukemia induce transformation of 32D cells mediated by the Ras and STAT5 pathways
  Blood, 2000
• Essential Role of Signal Transducer and Activator of Transcription (Stat)5a but Not Stat5b for Flt3-Dependent Signaling
  The Journal of Experimental Medicine, 2000
• The role of STATs in myeloid differentiation and leukemia
  Oncogene, 2000
• Tandem-duplicated Flt3 constitutively activates STAT5 and MAP kinase and introduces autonomous cell growth in IL-3-dependent cell lines
  Oncogene, 2000
• Detection of Intracellular Phosphorylated STAT-1 by Flow Cytometry
  Clinical Immunology, 1999
• Constitutive activation of STAT proteins in primary lymphoid and myeloid leukemia cells and in Epstein-Barr virus (EBV)-related lymphoma cell lines
  Blood, 1996
• Tyrosyl phosphorylation and DNA binding activity of signal transducers and activators of transcription (STAT) proteins in hematopoietic cell lines transformed by Bcr/Abl.
  The Journal of Experimental Medicine, 1996