Possible Involvement of a Central Noradrenergic System in Automutilation Induced by Clonidine in Mice
Open Access
- 1 January 1977
- journal article
- research article
- Published by Elsevier in The Japanese Journal of Pharmacology
- Vol. 27 (1) , 145-152
- https://doi.org/10.1254/jjp.27.145
Abstract
Japan's largest platform for academic e-journals: J-STAGE is a full text database for reviewed academic papers published by Japanese societiesThis publication has 21 references indexed in Scilit:
- Effects of phenoxybenzamine, aceperone and clonidine on the level of 3-methoxy-4-hydroxyphenylglycol (MOPEG) in rat brainJournal of Pharmacy and Pharmacology, 1974
- Sensitization by caffeine of central catecholamine receptorsJournal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1973
- The effect of clonidine on locomotor activity in miceLife Sciences, 1972
- Drug‐Induced Changes in the Release of3H‐Monoamines from Field Stimulated Rat Brain SlicesActa Physiologica Scandinavica, 1971
- Studies on the action of some psychoactive drugs on central noradrenaline neurones after inhibition of dopamine-β-hydroxylaseBrain Research, 1970
- Evidence for a central noradrenaline receptor stimulation by clonidineLife Sciences, 1970
- Aggressive behavior induced by large doses of 2-(2,6-dichlorphenylamino)-2-imidazoline hydrochloride (ST 155) in miceEuropean Journal of Pharmacology, 1968
- Centrally mediated decrease in sympathetic tone induced by 2(2,6-dichlorophenylamino)-2 imidazoline (S.T. 155, Catapresan)European Journal of Pharmacology, 1967
- Acute hypotensive action of 2-(2,6-dichlorophenylamino)- 2-imidazoline hydrochloride (St 155) after infusion into the cat's vertebral arteryEuropean Journal of Pharmacology, 1967
- A sensitive method for spectrophotofluorometric assay of catecholaminesInternational Journal of Neuropharmacology, 1964