Loss of functional KATP channels in pancreatic β–cells causes persistent hyperinsulinemic hypoglycemia of infancy

This publication has 19 references indexed in Scilit:

• Evaluation of fasts for investigating hypoglycaemia or suspected metabolic disease.
  Archives of Disease in Childhood, 1996
• Cloning and functional expression of the cDNA encoding a novel ATP‐sensitive potassium channel subunit expressed in pancreatic β‐cells, brain, heart and skeletal muscle
  FEBS Letters, 1995
• Mutations in the Sulfonylurea Receptor Gene in Familial Persistent Hyperinsulinemic Hypoglycemia of Infancy
  Science, 1995
• Familial hyperinsulinism maps to chromosome 11p14–15.1, 30 cM centromeric to the insulin gene
  Nature Genetics, 1994
• Mutations in CFTR associated with mild-disease-form CI- channels with altered pore properties
  Nature, 1993
• Surgical treatment of hyperinsulinaemic hypoglycaemia in infancy and childhood.
  Archives of Disease in Childhood, 1992
• Nesidioblastosis: evidence for autosomal recessive inheritance.
  Archives of Disease in Childhood, 1991
• Potassium selective ion channels in insulin-secreting cells: physiology, pharmacology and their role in stimulus-secretion coupling
  Biochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1991
• Regulation of insulin release in persistent hyperinsulinaemic hypoglycaemia of infancy studied in long-term culture of pancreatic tissue
  Diabetologia, 1990
• Nesidioblastosis of the pancreas: definition of the syndrome and the management of the severe neonatal hyperinsulinaemic hypoglycaemia.
  Archives of Disease in Childhood, 1981