Expression of phosphorylated c-Jun N-terminal protein kinase (JNK) in experimental glaucoma in rats
- 30 April 2006
- journal article
- Published by Elsevier in Experimental Eye Research
- Vol. 82 (4) , 576-582
- https://doi.org/10.1016/j.exer.2005.08.017
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- CNTF promotes survival of retinal ganglion cells after induction of ocular hypertension in rats: the possible involvement of STAT3 pathwayEuropean Journal of Neuroscience, 2004
- A peptide inhibitor of c-Jun N-terminal kinase protects against excitotoxicity and cerebral ischemiaNature Medicine, 2003
- Retinal Ganglion Cell Protection with Geranylgeranylacetone, a Heat Shock Protein Inducer, in a Rat Glaucoma ModelInvestigative Opthalmology & Visual Science, 2003
- Differential activation of MAPK/ERK and p38/SAPK in neurones and glia following focal cerebral ischaemia in the ratMolecular Brain Research, 2000
- Translocation of SAPK/JNK to Mitochondria and Interaction with Bcl-xL in Response to DNA DamageJournal of Biological Chemistry, 2000
- Expression of cell death-associated phospho-c-Jun and p53-activated gene 608 in hippocampal CA1 neurons following global ischemiaMolecular Brain Research, 1999
- MEK1 protein kinase inhibition protects against damage resulting from focal cerebral ischemiaProceedings of the National Academy of Sciences, 1999
- Lasting N-Terminal Phosphorylation of c-Jun and Activation of c-Jun N-Terminal Kinases after Neuronal InjuryJournal of Neuroscience, 1998
- Signal transduction by the c-Jun N-terminal kinase (JNK) — from inflammation to developmentCurrent Opinion in Cell Biology, 1998
- TUNEL-Positive Ganglion Cells in Human Primary Open-angle GlaucomaArchives of Ophthalmology (1950), 1997