Differential Binding of the NFE3 and CP1/NFY Transcription Factors to the Human γ- and ∊-Globin CCAAT Boxes
Open Access
- 1 September 1995
- journal article
- research article
- Published by Elsevier
- Vol. 270 (37) , 21934-21941
- https://doi.org/10.1074/jbc.270.37.21934
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- A single point mutation is the cause of the Greek form of hereditary persistence of fetal haemoglobinNature, 1992
- Treatment of advanced chronic lymphocytic leukemia by fludarabineAnnals of Hematology, 1991
- Human γ-globin genes silenced independently of other genes in the β-globin locusNature, 1991
- Autonomous Developmental Control of Human Embryonic Globin Gene Switching in Transgenic MiceScience, 1990
- Tandem AP-1-binding sites within the human beta-globin dominant control region function as an inducible enhancer in erythroid cells.Genes & Development, 1990
- Increased γ-globin expression in a nondeletion HPFH mediated by an erythroid-specif ic DNA-binding factorNature, 1989
- Position-independent, high-level expression of the human β-globin gene in transgenic miceCell, 1987
- A nuclear factor that binds to a conserved sequence motif in transcriptional control elements of immunoglobulin genesNature, 1986
- A point mutation in the Aγ-globin gene promoter in Greek hereditary persistence of fetal haemoglobinNature, 1985
- G to A substitution in the distal CCAAT box of the Aγ-globin gene in Greek hereditary persistence of fetal haemoglobinNature, 1985