Dichloroacetate and Trichloroacetate Promote Clonal Expansion of Anchorage-Independent Hepatocytesin Vivoandin Vitro
- 30 June 1998
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 150 (2) , 287-294
- https://doi.org/10.1006/taap.1998.8417
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- The carcinogenicity of dichloroacetic acid in the male fischer 344 ratToxicology, 1996
- Biochemical, pathologic and morphometric alterations induced in male B6C3F1 mouse liver by short-term exposure to dichloroacetic acidToxicology Letters, 1995
- Negative selection in hepatic tumor promotion in relation to cancer risk assessmentToxicology, 1995
- Dichloroacetic acid and related compounds: induction of prophage in E. coli and mutagenicity and mutation spectra in Salmonella TA100Mutagenesis, 1994
- Differential transformation efficiency but not AP-1 induction under anchorage-dependent and -independent conditionsCarcinogenesis: Integrative Cancer Research, 1994
- Mechanisms of multistep carcinogenesis and carcinogen risk assessment.Environmental Health Perspectives, 1993
- Analysis of DNA strand breaks induced in rodent liver in vivo, hepatocytes in primary culture, and a human cell line by chlorinated acetic acids and chlorinated acetaldehydesEnvironmental and Molecular Mutagenesis, 1992
- The carcinogenicity of dichloroacetic acid in the male B6C3F1 mouseFundamental and Applied Toxicology, 1991
- Liver tumor induction in B6C3F1 mice by dichloroacetate and trichloroacetateToxicology, 1990
- Tumour promoter induces anchorage independence irreversiblyNature, 1979