ER stress contributes to ischemia-induced cardiomyocyte apoptosis
- 12 September 2006
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 349 (4) , 1406-1411
- https://doi.org/10.1016/j.bbrc.2006.09.009
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Activation of endoplasmic reticulum stress response during the development of ischemic heart diseaseAmerican Journal of Physiology-Heart and Circulatory Physiology, 2006
- Mediators of endoplasmic reticulum stress‐induced apoptosisEMBO Reports, 2006
- AMP-Activated Protein Kinase Protects Cardiomyocytes against Hypoxic Injury through Attenuation of Endoplasmic Reticulum StressMolecular and Cellular Biology, 2005
- Functional and clinical repercussions of myocyte apoptosis in the multifaceted damage by ischemia/reperfusion injury: old and new concepts after 10 years of contributionsCell Death & Differentiation, 2004
- Activating Transcription Factor 4 Is Translationally Regulated by Hypoxic StressMolecular and Cellular Biology, 2004
- Oxidative injury to the endoplasmic reticulum in mouse brains after transient focal ischemiaNeurobiology of Disease, 2004
- XBP-1 Regulates a Subset of Endoplasmic Reticulum Resident Chaperone Genes in the Unfolded Protein ResponseMolecular and Cellular Biology, 2003
- Overexpression of the stress‐protein Grp94 reduces cardiomyocyte necrosis due to calcium overload and simulated ischemiaThe FASEB Journal, 2003
- Electrophysiologic Changes in Ischemic Ventricular Myocardium: I. Influence of Ionic, Metabolic, and Energetic ChangesJournal of Cardiovascular Electrophysiology, 1995
- MYOCARDIAL METABOLISM AND HEART DISEASEJapanese Circulation Journal, 1978