Angiotensin-converting enzyme gene I/D polymorphism and carotid artery disease in renovascular hypertension
Open Access
- 1 February 2000
- journal article
- research article
- Published by Oxford University Press (OUP)
- Vol. 13 (2) , 128-133
- https://doi.org/10.1016/s0895-7061(99)00186-7
Abstract
There is evidence linking the activation of the renin-angiotensin system (RAS) with target organ damage in renovascular hypertension (RVH). A geneticKeywords
This publication has 22 references indexed in Scilit:
- Comparison of Target Organ Damage in Renovascular and Essential HypertensionPublished by Oxford University Press (OUP) ,1996
- 1995 Update of the Working Group Reports on Chronic Renal Failure and Renovascular HypertensionArchives of internal medicine (1960), 1996
- Association between a Deletion Polymorphism of the Angiotensin-Converting-Enzyme Gene and Left Ventricular HypertrophyNew England Journal of Medicine, 1994
- Deletion polymorphism in the gene for angiotensin-converting enzyme is a potent risk factor for myocardial infarctionNature, 1992
- Effect of Captopril on Mortality and Morbidity in Patients with Left Ventricular Dysfunction after Myocardial InfarctionNew England Journal of Medicine, 1992
- Blood pressure-independent effect of angiotensin inhibition on vascular lesions of chronic renal failureKidney International, 1992
- Simplified captopril renography in diagnosis and treatment of renal artery stenosis.Hypertension, 1991
- An insertion/deletion polymorphism in the angiotensin I-converting enzyme gene accounting for half the variance of serum enzyme levels.Journal of Clinical Investigation, 1990
- Two putative active centers in human angiotensin I-converting enzyme revealed by molecular cloning.Proceedings of the National Academy of Sciences, 1988
- Detection, Evaluation, and Treatment of Renovascular HypertensionArchives of internal medicine (1960), 1987