Developmental expression of the GIRK family of inward rectifying potassium channels: implications for abnormalities in the weaver mutant mouse
- 19 December 1997
- journal article
- Published by Elsevier in Brain Research
- Vol. 778 (2) , 251-264
- https://doi.org/10.1016/s0006-8993(97)00896-2
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Heteromeric channel formation and Ca2+‐free media reduce the toxic effect of the weaver Kir 3.2 alleleFEBS Letters, 1996
- The weaver mutation changes the ion selectivity of the affected inwardly rectifying potassium channel GIRK2FEBS Letters, 1996
- Functional Analysis of the weaver Mutant GIRK2 K+ Channel and Rescue of weaver Granule CellsNeuron, 1996
- A potassium channel mutation in weaver mice implicates membrane excitability in granule cell differentiationNature Genetics, 1995
- Heterologous Multimeric Assembly Is Essential for K+ Channel Activity of Neuronal and Cardiac G-Protein-Activated Inward RectifiersBiochemical and Biophysical Research Communications, 1995
- Molecular Cloning of a Mouse G-Protein-Activated K+ Channel (mGIRK1) and Distinct Distributions of 3 GIRK (GIRK1, 2 and 3) mRNAs in Mouse BrainBiochemical and Biophysical Research Communications, 1995
- Cloning provides evidence for a family of inward rectifier and G‐protein coupled K+ channels in the brainFEBS Letters, 1994
- Male-sterile phenotype of the neurological mouse mutant weaverDevelopmental Dynamics, 1994
- Purkinje cell loss is due to a direct action of the weaver gene in Purkinje cells: evidence from chimeric miceDevelopmental Brain Research, 1990
- The weaver granuloprival phenotype is due to intrinsic action of the mutant locus in granule cells: Evidence from homozygous weaver chimerasNeuron, 1989