Termination of pseudopregnancy in the rat produces an anxiogenic-like response that is associated with an increase in benzodiazepine receptor binding density and a decrease in GABA-stimulated chloride influx in the hippocampus
- 30 January 2005
- journal article
- research article
- Published by Elsevier in Brain Research Bulletin
- Vol. 64 (6) , 511-518
- https://doi.org/10.1016/j.brainresbull.2004.11.001
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Tolerance to the anticonvulsant activity of midazolam and allopregnanolone in a model of picrotoxin seizuresEuropean Journal of Pharmacology, 2001
- Activation of peripheral mitochondrial benzodiazepine receptors in the hippocampus stimulates allopregnanolone synthesis and produces anxiolytic-like effects in the ratPsychopharmacology, 2000
- Anxiolytic effects of the neuroactive steroid pregnanolone (3α-OH-5β-pregnan-20-one) after microinjection in the dorsal hippocampus and lateral septumBrain Research, 1999
- Bidirectional Alterations of GABAA Receptor Subunit Peptide Levels in Rat Cortex During Chronic Ethanol Consumption and WithdrawalJournal of Neurochemistry, 1997
- The anxiolytic-like effects of the neurosteroid allopregnanolone: interactions with GABAA receptorsEuropean Journal of Pharmacology, 1997
- Withdrawal from the endogenous steroid progesterone results in GABAA currents insensitive to benzodiazepine modulation in rat CA1 hippocampusJournal of Neurophysiology, 1995
- Anxiolytic Effect of Progesterone is Mediated by the Neurosteroid Allopregnanolone at Brain GABAA ReceptorsJournal of Neuroendocrinology, 1995
- Expression of high density lipoprotein-binding protein messenger ribonucleic acid in the rat ovary and its regulation by gonadotropinEndocrinology, 1994
- Anxiolytic effects of 3α-hydroxy-5α[β]-pregnan-20-one: endogenous metabolites of progesterone that are active at the GABAA receptorBrain Research, 1991
- Anticonvulsant profile of the progesterone metabolite 5α-pregnan-3α-ol-20-oneEuropean Journal of Pharmacology, 1989