Immunosuppressive and Proinflammatory Activities of the VacA Toxin of Helicobacter pylori
Open Access
- 15 December 2003
- journal article
- editorial
- Published by Rockefeller University Press in The Journal of Experimental Medicine
- Vol. 198 (12) , 1767-1771
- https://doi.org/10.1084/jem.20031839
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Selective increase of the permeability of polarized epithelial cell monolayers by Helicobacter pylori vacuolating toxin.Journal of Clinical Investigation, 1998
- The Acid Activation ofHelicobacter pyloriToxin VacA: Structural and Membrane Binding StudiesBiochemical and Biophysical Research Communications, 1998
- Persistent Infection byHelicobacter pyloriDown‐Modulates Virus‐Specific CD8+Cytotoxic T Cell Response and Prolongs Viral InfectionThe Journal of Infectious Diseases, 1998
- TRANSCRIPTION FACTORS OF THE NFAT FAMILY:Regulation and FunctionAnnual Review of Immunology, 1997
- The vacuolar ATPase proton pump is present on intracellular vacuoles induced by Helicobacter pyloriJournal of Medical Microbiology, 1996
- Tuberculosis into the next century: Proceedings of a symposium held on 4 February 1995 at the Liverpool School of Tropical MedicineJournal of Medical Microbiology, 1996
- Lipid Interaction of the 37‐kDa and 58‐kDa Fragments of the Helicobacter Pylori CytotoxinEuropean Journal of Biochemistry, 1995
- Cdc42 and PAK-mediated Signaling Leads to Jun Kinase and p38 Mitogen-activated Protein Kinase ActivationJournal of Biological Chemistry, 1995
- Rho Family GTPases Regulate p38 Mitogen-activated Protein Kinase through the Downstream Mediator Pak1Journal of Biological Chemistry, 1995
- The small GTP-binding proteins Rac1 and Cdc42regulate the activity of the JNK/SAPK signaling pathwayCell, 1995