Overexpression of a Modified Human Malonyl-CoA Decarboxylase Blocks the Glucose-induced Increase in Malonyl-CoA Level but Has No Impact on Insulin Secretion in INS-1-derived (832/13) β-Cells
Open Access
- 1 March 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (9) , 6479-6484
- https://doi.org/10.1074/jbc.m010364200
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- A Role for Malonyl-CoA in Glucose-Stimulated Insulin Secretion from Clonal Pancreatic β-CellsPublished by Elsevier ,2021
- Acute Stimulation with Long Chain Acyl-CoA Enhances Exocytosis in Insulin-secreting Cells (HIT T-15 and NMRI β-Cells)Journal of Biological Chemistry, 2000
- The Role of Long-Chain Fatty Acyl-CoA Esters in β-Cell Signal TransductionJournal of Nutrition, 2000
- Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose.Diabetes, 1999
- Molecular or Pharmacologic Perturbation of the Link between Glucose and Lipid Metabolism Is without Effect on Glucose-stimulated Insulin SecretionPublished by Elsevier ,1998
- Glucose stimulation of insulin release in the absence of extracellular Ca2+ and in the absence of any increase in intracellular Ca2+ in rat pancreatic islets.Proceedings of the National Academy of Sciences, 1995
- METABOLIC COUPLING FACTORS IN PANCREATIC β-CELL SIGNAL TRANSDUCTIONAnnual Review of Biochemistry, 1995
- Evidence that glucose can control insulin release independently from its action on ATP-sensitive K+ channels in mouse B cells.Journal of Clinical Investigation, 1992
- Malonyl-CoA and long chain acyl-CoA esters as metabolic coupling factors in nutrient-induced insulin secretion.Journal of Biological Chemistry, 1992