The Activity of the Epithelial Sodium Channel Is Regulated by Clathrin-mediated Endocytosis
Open Access
- 1 October 1997
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 272 (41) , 25537-25541
- https://doi.org/10.1074/jbc.272.41.25537
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Cell surface expression of the epithelial Na channel and a mutant causing Liddle syndrome: A quantitative approachProceedings of the National Academy of Sciences, 1996
- Liddle disease caused by a missense mutation of beta subunit of the epithelial sodium channel gene.Journal of Clinical Investigation, 1996
- Peptide block of constitutively activated Na+ channels in Liddle's diseaseAmerican Journal of Physiology-Cell Physiology, 1996
- A de novo missense mutation of the beta subunit of the epithelial sodium channel causes hypertension and Liddle syndrome, identifying a proline-rich segment critical for regulation of channel activity.Proceedings of the National Academy of Sciences, 1995
- Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channelCell, 1995
- Renal sodium channels: Regulation and single channel propertiesKidney International, 1995
- Hypertension caused by a truncated epithelial sodium channel γ subunit: genetic heterogeneity of Liddle syndromeNature Genetics, 1995
- A mutation in the epithelial sodium channel causing Liddle disease increases channel activity in the Xenopus laevis oocyte expression system.Proceedings of the National Academy of Sciences, 1995
- Liddle's syndrome: Heritable human hypertension caused by mutations in the β subunit of the epithelial sodium channelCell, 1994
- A general method of site-specific mutagenesis using a modification of the Thermus aquaticus polymerase chain reactionAnalytical Biochemistry, 1989