Potentiation of Excitotoxicity in Transgenic Mice Overexpressing Neuronal Cyclooxygenase-2
- 1 September 1999
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 155 (3) , 995-1004
- https://doi.org/10.1016/s0002-9440(10)65199-1
Abstract
No abstract availableKeywords
Funding Information
- National Institute on Aging (AG14766, AG13799, AG14239)
This publication has 52 references indexed in Scilit:
- DNA Fragmentation and Prolonged Expression of c-fos, c-jun, and hsp70 in Kainic Acid-Induced Neuronal Cell Death in Transgenic Mice Overexpressing Human CuZn-Superoxide DismutaseJournal of Cerebral Blood Flow & Metabolism, 1997
- Regulatory Functions of Phospholipase A2Critical Reviews in Immunology, 1997
- Self-Promotion? Intimate Connections Between APC and Prostaglandin H Synthase-2Cell, 1996
- Lack of β-Amyloidosis in transgenic mice expressing low levels of familial Alzheimer's disease missense mutationsNeurobiology of Aging, 1996
- DBL Expression Driven by the Neuron Specific Enolase Promoter Induces Tumor Formation in Transgenic Mice with a P53(±) Genetic BackgroundBiochemical and Biophysical Research Communications, 1995
- A signal terminatorNature, 1995
- Reduced Risk of Large-Bowel Adenomas Among Aspirin UsersJNCI Journal of the National Cancer Institute, 1993
- Structure, chromosome mapping and regulation of the mouse zinc-finger gene Krox-24; evidence for a common regulatory pathway for immediate-early serum-response genesGene, 1989
- Glutamate toxicity in a neuronal cell line involves inhibition of cystine transport leading to oxidative stressNeuron, 1989
- The effect of incomplete cerebral ischemia on prostaglandin levels in rat brain.Stroke, 1982