Genetic deficiency or pharmacological inhibition of cyclooxygenase‐1 or ‐2 induces mouse keratinocyte differentiation in vitro and in vivo
Open Access
- 3 November 2003
- journal article
- Published by Wiley in The FASEB Journal
- Vol. 18 (1) , 185-187
- https://doi.org/10.1096/fj.02-1192fje
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Inhibitory effects of mofezolac, a cyclooxygenase-1 selective inhibitor, on intestinal carcinogenesisCarcinogenesis: Integrative Cancer Research, 2002
- Why there are two cyclooxygenase isozymesJournal of Clinical Investigation, 2001
- Abnormal differentiation of epidermis in transgenic mice constitutively expressing cyclooxygenase-2 in skinProceedings of the National Academy of Sciences, 2001
- Reduction of UV-induced skin tumors in hairless mice by selective COX-2 inhibitionCarcinogenesis: Integrative Cancer Research, 1999
- A Selective Cyclooxygenase‐2 Inhibitor Suppresses Tumor Growth in Nude Mouse Xenografted with Human Head and Neck Squamous Carcinoma CellsJapanese Journal of Cancer Research, 1999
- Chemopreventive activity of celecoxib, a specific cyclooxygenase-2 inhibitor, and indomethacin against ultraviolet light-induced skin carcinogenesisMolecular Carcinogenesis, 1999
- Localization of prostaglandin H synthase isoenzymes in murine epidermal tumors: Suppression of skin tumor promotion by inhibition of prostaglandin H synthase-2Molecular Carcinogenesis, 1998
- Localization of Prostaglandin-H Synthase-1 and -2 in Mouse Skin: Implications for Cutaneous FunctionExperimental Cell Research, 1998
- Prostaglandin Endoperoxide H Synthases-1 and -2Published by Elsevier ,1996
- Differential expression of prostaglandin-H synthase isoenzymes in normal and activated keratinocytes in vivo and in vitroBiochemical Journal, 1995