WHY CORTICOIDS ARE EFFECTIVE AGAINST A WIDE VARIETY OF CANCERS
- 1 March 1974
- journal article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 230 (1) , 489-490
- https://doi.org/10.1111/j.1749-6632.1974.tb14483.x
Abstract
No abstract availableThis publication has 10 references indexed in Scilit:
- The Problem of PainNature, 1973
- EVIDENCE THAT THE BONE RESORPTION-STIMULATING FACTOR PRODUCED BY MOUSE FIBROSARCOMA CELLS IS PROSTAGLANDIN E2The Journal of Experimental Medicine, 1972
- Glucocorticoid Action on Hybrid Clones Derived from Cultured Myeloma and Lymphoma Cell LinesProceedings of the National Academy of Sciences, 1972
- Stereospecific Glucocorticoid Binding to Subcellular Fractions of the Sensitive and Resistant Lymphosarcoma P1798Nature New Biology, 1971
- Specific Cytoplasmic Glucocorticoid Hormone Receptors in Hepatoma Tissue Culture CellsProceedings of the National Academy of Sciences, 1971
- Glucocorticoid Receptors in Lymphoma Cells in Culture: Relationship to Glucocorticoid Killing ActivityScience, 1971
- The Relationship between Glucocorticoid Binding and Tyrosine Aminotransferase Induction in Hepatoma Tissue Culture CellsProceedings of the National Academy of Sciences, 1970
- Hormone Induced Modification of the Cell SurfaceNature, 1969
- Chemical Suppression of Adaptive ImmunityPublished by Elsevier ,1967
- binding of cortisol-1, 2-3H by a substance in the supernatant fraction of P1798 mouse lymphosarcomaBiochemical and Biophysical Research Communications, 1966