Role of K+ channels and sodium pump in the vasodilation induced by acetylcholine, nitric oxide, and cyclic GMP in the rabbit aorta
- 7 July 1999
- journal article
- Published by Elsevier in General Pharmacology: The Vascular System
- Vol. 33 (1) , 35-41
- https://doi.org/10.1016/s0306-3623(98)00259-6
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Role of Potassium Channels in Cerebral Blood VesselsStroke, 1995
- NG-nitro-l-arginine-resistant endothelium-dependent relaxation induced by acetylcholine in the rabbit renal arteryLife Sciences, 1994
- Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscleNature, 1994
- Comparison of the actions of acetylcholine and BRL 38227 in the guinea‐pig coronary arteryBritish Journal of Pharmacology, 1992
- Evidence that nitric oxide does not mediate the hyperpolarization and relaxation to acetylcholine in the rat small mesenteric arteryBritish Journal of Pharmacology, 1992
- Endothelium‐dependent and BRL 34915‐induced vasodilatation in rat isolated perfused mesenteric arteries: role of G‐proteins, K+ and calcium channelsBritish Journal of Pharmacology, 1990
- Endothelium‐dependent relaxation and hyperpolarization of canine coronary artery smooth muscles in relation to the electrogenic Na‐K pumpBritish Journal of Pharmacology, 1989
- Acetylcholine releases endothelium‐derived hyperpolarizing factor and EDRF from rat blood vesselsBritish Journal of Pharmacology, 1988
- Endothelium‐dependent hyperpolarization of canine coronary smooth muscleBritish Journal of Pharmacology, 1988
- The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholineNature, 1980