Somnolence, akinesia, and sensory activation of motivated behavior in the lateral hypothalamic syndrome.
- 1 July 1975
- journal article
- Published by Proceedings of the National Academy of Sciences in Proceedings of the National Academy of Sciences
- Vol. 72 (7) , 2819-2823
- https://doi.org/10.1073/pnas.72.7.2819
Abstract
After lateral hypothalamic damage in rats, somnolence, akinesia, and sensory neglect combine to produce complete aphagia. Only simple automatisms (such as grooming, chewing, licking) are present, but intense stimuli can activate more complex actions (walking, orientation, swimming). In the anorexic stage, tactile stimuli dominate in steering locomotion and "spontaneous" locomotion depends on activation from the empty stomach.Keywords
This publication has 31 references indexed in Scilit:
- Further analysis of sensory inattention following lateral hypothalamic damage in rats.Journal of Comparative and Physiological Psychology, 1974
- Development of Grooming in Mice with Amputated ForelimbsScience, 1973
- Sensorimotor syndrome produced by lesions of the amygdala and lateral hypothalamus.Journal of Comparative and Physiological Psychology, 1973
- Limbic-diencephalic mechanisms of voluntary movement.Psychological Review, 1971
- Starvation retards development of food and water regulations.Journal of Comparative and Physiological Psychology, 1971
- Relationship of body weight to the lateral hypothalamic feeding syndrome.Journal of Comparative and Physiological Psychology, 1970
- STAGES OF RECOVERY AND DEVELOPMENT OF LATERAL HYPOTHALAMIC CONTROL OF FOOD AND WATER INTAKEAnnals of the New York Academy of Sciences, 1969
- Exaggerated prandial drinking in the "recovered lateral" rat without saliva.Journal of Comparative and Physiological Psychology, 1969
- An irritative hypothesis concerning the hypothalamic regulation of food intake.Psychological Review, 1965
- Recovery from the Failure to Eat Produced by Hypothalamic LesionsScience, 1954