Smads 2 and 3 Are Differentially Activated by Transforming Growth Factor-β (TGF-β) in Quiescent and Activated Hepatic Stellate Cells

This publication has 68 references indexed in Scilit:

• Expression of Smads during in Vitro Transdifferentiation of Hepatic Stellate Cells to Myofibroblasts
  Biochemical and Biophysical Research Communications, 2001
• Interaction between Smad Anchor for Receptor Activation and Smad3 Is Not Essential for TGF-β/Smad3-Mediated Signaling
  Biochemical and Biophysical Research Communications, 2001
• Two Divergent Signaling Pathways for TGF-β Separated by a Mutation of Its Type II Receptor Gene
  Biochemical and Biophysical Research Communications, 1999
• Role of Transforming Growth Factor β Type Ii Receptor in Hepatic Fibrosis: Studies of Human Chronic Hepatitis C and Experimental Fibrosis in Rats
  Hepatology, 1999
• The role of TGFβ1 in initiating hepatic stellate cell activation in vivo
  Published by Elsevier ,1999
• Expression patterns of matrix metalloproteinases and their inhibitors in parenchymal and non-parenchymal cells of rat liver: regulation by TNF-α and TGF-β1
  Journal of Hepatology, 1999
• Localization and cellular sources of activins in normal and fibrotic rat liver
  Hepatology, 1997
• Transforming growth factor β-induced phosphorylation of Smad3 is required for growth inhibition and transcriptional induction in epithelial cells
  Proceedings of the National Academy of Sciences, 1997
• Tumor necrosis factor alpha (TNFα) and transforming growth factor β1 (TGFβ1) stimulate fibronectin synthesis and the transdifferentiation of fat-storing cells in the rat liver into myofibroblasts
  Virchows Archiv B Cell Pathology Including Molecular Pathology, 1993
• Metabolism and effects on biliary lipid secretion of murocholic acid in the hamster
  Journal of Hepatology, 1990