Development of Insulitis and Diabetes in B Cell-Deficient NOD Mice
- 30 June 1997
- journal article
- Published by Elsevier in Journal of Autoimmunity
- Vol. 10 (3) , 257-260
- https://doi.org/10.1006/jaut.1997.0128
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- B lymphocytes are essential for the initiation of T cell-mediated autoimmune diabetes: analysis of a new "speed congenic" stock of NOD.Ig mu null mice.The Journal of Experimental Medicine, 1996
- Genetic Control of Autoimmune Diabetes in the Nod MouseAnnual Review of Immunology, 1995
- Failure of a protective major histocompatibility complex class II molecule to delete autoreactive T cells in autoimmune diabetes.Proceedings of the National Academy of Sciences, 1993
- Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic miceNature, 1993
- Spontaneous loss of T-cell tolerance to glutamic acid decarboxylase in murine insulin-dependent diabetesNature, 1993
- Adoptive Transfer of Diabetes Into Immunodeficient NOD-scid/scid Mice: Relative Contributions of CD4+ and CD8+ T-Cells From Diabetic Versus Prediabetic NOD.NON-Thy-1a DonorsDiabetes, 1993
- A polyalanine peptide with only five native myelin basic protein residues induces autoimmune encephalomyelitis.The Journal of Experimental Medicine, 1992
- A B cell-deficient mouse by targeted disruption of the membrane exon of the immunoglobulin μ chain geneNature, 1991
- Identification of the 64K autoantigen in insulin-dependent diabetes as the GABA-synthesizing enzyme glutamic acid decarboxylaseNature, 1990
- The natural history of lymphocyte subsets infiltrating the pancreas of NOD miceDiabetologia, 1989