Possible epigenetic mechanisms of tumor progression: Induction of high-frequency heritable but phenotypically unstable changes in the tumorigenic and metastatic properties of tumor cell populations by 5-azacytidine treatment
- 1 January 1984
- journal article
- review article
- Published by Wiley in Journal of Cellular Physiology
- Vol. 121 (S3) , 87-97
- https://doi.org/10.1002/jcp.1041210411
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Two DNA methyltransferases from murine erythroleukemia cells: purification, sequence specificity, and mode of interaction with DNA.Proceedings of the National Academy of Sciences, 1983
- Specific gene mutations in L5178Y cells in culture: A report of the U.S. environmental protection agency gene-tox programMutation Research/Reviews in Genetic Toxicology, 1983
- DNA METHYLATION AND GENE ACTIVITYAnnual Review of Biochemistry, 1983
- Antigenic Tumor Cell Variants Obtained with MutagensAdvances in Cancer Research, 1983
- Induction of sister chromatid exchanges by 5-fluorodeoxycytidine: correlation with DNA methylationChromosoma, 1982
- Mutagenic responses of five independent geentic loci in CHO cells to a variety of mutagensMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1982
- Mutagenesis by chemical agents in V79 Chinese hamster cells: A review and analysis of the literature: A report of the gene-tox programMutation Research/Reviews in Genetic Toxicology, 1981
- A new theory of carcinogenesisBritish Journal of Cancer, 1979
- Rejection by syngeneic mice of cell variants obtained by mutagenesis of a malignant teratocarcinoma cell line.Proceedings of the National Academy of Sciences, 1977
- Biological Effects of 5-Azacytidine in EukaryotesOncology, 1974