Abstract
The cerebral palsies of childhood have been intensively studied clinically and have been divided into a number of fairly characteristic categories.1Knowledge of the pathology of this group of common neurologic conditions has not kept pace with clinical studies. Very few patients die in the acute stages of the disease. Most of the material obtained at necropsy therefore consists rather of the scars than of the causal lesions. Analysis of the changes observed has failed to produce a clear conception of the pathologic basis of the condition, its etiology and pathogenesis. Since the address of Strümpell2before the German Society of Naturalists and Physicians in 1884, the concept of acute encephalitis in early childhood as a common cause of infantile cerebral paralysis has been received with wide interest in neurologic and pediatric circles. Strümpell noted a close analogy of the condition to infantile spinal paralysis, both being acute

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