CD95 (Fas/APO-1) Induces Ceramide Formation and Apoptosis in the Absence of a Functional Acid Sphingomyelinase

This publication has 41 references indexed in Scilit:

• Tumor Necrosis Factor α Activates NF-κB in Acid Sphingomyelinase-deficient Mouse Embryonic Fibroblasts
  Published by Elsevier ,1997
• Apoptosis by Death Factor
  Cell, 1997
• Ceramide induces apoptosis via CPP32 activation
  FEBS Letters, 1996
• FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex
  Cell, 1996
• Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death
  Cell, 1996
• A Novel Cytoplasmic Domain of the p55 Tumor Necrosis Factor Receptor Initiates the Neutral Sphingomyelinase Pathway
  Journal of Biological Chemistry, 1996
• Molecular Ordering of the Cell Death Pathway: Bcl-2 AND Bcl-xL FUNCTION UPSTREAM OF THE CED-3-LIKE APOPTOTIC PROTEASES
  Published by Elsevier ,1996
• CPP32/Apopain Is a Key Interleukin 1β Converting Enzyme-like Protease Involved in Fas-mediated Apoptosis
  Journal of Biological Chemistry, 1996
• A novel domain within the 55 kd TNF receptor signals cell death
  Cell, 1993
• Programmed Cell Death Induced by Ceramide
  Science, 1993