Chronic Brain Oxidation in a Glutathione Peroxidase Knockout Mouse Model Results in Increased Resistance to Induced Epileptic Seizures
- 31 August 2000
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 164 (2) , 257-268
- https://doi.org/10.1006/exnr.2000.7431
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Limbic seizure and brain damage produced by kainic acid: Mechanisms and relevance to human temporal lobe epilepsyPublished by Elsevier ,2003
- Subunit Composition ofN-Methyl-d-aspartate Receptors in the Central Nervous System that Contain the NR2D SubunitMolecular Pharmacology, 1998
- Constitutive overexpression of Cu/Zn superoxide dismutase exacerbates kainic acid-induced apoptosis of transgenic-Cu/Zn superoxide dismutase neurons.Proceedings of the National Academy of Sciences, 1996
- A cautionary note on the use of the TUNEL stain to determine apoptosisNeuroReport, 1995
- Age-related alterations of NMDA-receptor properties in the mouse forebrain: partial restoration by chronic phosphatidylserune treatmentBrain Research, 1992
- Oxygen free radicals regulate NMDA receptor function via a redox modulatory siteNeuron, 1990
- Ketamine, Phencyclidine, and MK‐801 Protect Against Kainic Acid‐Induced Seizure‐Related Brain DamageEpilepsia, 1990
- The action of hydrogen peroxide on paired pulse and long-term potentiation in the hippocampusFree Radical Biology & Medicine, 1989
- Changes in minor transcripts from the α1 and βmajglobin and glutathione peroxidase genes during erythropoiesisNucleic Acids Research, 1987
- [12] Assays of glutathione peroxidasePublished by Elsevier ,1984