Abstract
Resistance in field isolates of E. graminis f. sp. hordei to the fungicide ethirimol is controlled, not by 1 major gene, but by a complex heritable system in which the genetic factors involved are primarily additive. Particularly favorable gene combinations established in resistant genotypes may be broken through recombination, leading to some breakdown of resistance. No evidence was obtained for linkage between resistance to ethirimol and 3 virulence genes in the pathogen. The implications of the use of ethirimol in disease control strategies are discussed.

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