OX40 Promotes Bcl-xL and Bcl-2 Expression and Is Essential for Long-Term Survival of CD4 T Cells
Top Cited Papers
- 1 September 2001
- Vol. 15 (3) , 445-455
- https://doi.org/10.1016/s1074-7613(01)00191-1
Abstract
No abstract availableKeywords
This publication has 67 references indexed in Scilit:
- Downregulation of Bim, a Proapoptotic Relative of Bcl-2, Is a Pivotal Step in Cytokine-Initiated Survival Signaling in Murine Hematopoietic ProgenitorsMolecular and Cellular Biology, 2001
- Expression of Bcl-XL Restores Cell Survival, but Not Proliferation and Effector Differentiation, in Cd28-Deficient T LymphocytesThe Journal of Experimental Medicine, 2000
- CD4 T cell traffic control:in vivo evidence that ligation of OX40 on CD4 T cells by OX40-ligand expressed on dendritic cells leads to the accumulation of CD4 T cells in B folliclesEuropean Journal of Immunology, 1999
- THE CENTRAL EFFECTORS OF CELL DEATH IN THE IMMUNE SYSTEMAnnual Review of Immunology, 1999
- MATURE T LYMPHOCYTE APOPTOSIS—Immune Regulation in a Dynamic and Unpredictable Antigenic EnvironmentAnnual Review of Immunology, 1999
- Interleukin-3-Induced Phosphorylation of BAD Through the Protein Kinase AktScience, 1997
- Accessory Molecule and Costimulation Requirements for CD4 T Cell ResponseCritical Reviews in Immunology, 1997
- Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell DeathCell, 1996
- bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell deathCell, 1993
- The Role of the CD28 Receptor During T Cell Responses to AntigenAnnual Review of Immunology, 1993