Exogenously administered interleukin-10 decreases pulmonary neutrophil infiltration in a tumor necrosis factor–dependent murine model of acute visceral ischemia
- 1 July 1997
- journal article
- Published by Elsevier in Journal of Vascular Surgery
- Vol. 26 (1) , 113-118
- https://doi.org/10.1016/s0741-5214(97)70154-x
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- VISCERAL ISCHEMIA-REPERFUSION INJURY PROMOTES TUMOR NECROSIS FACTOR (TNF) AND INTERLEUKIN-1 (IL-1) DEPENDENT ORGAN INJURY IN THE MOUSEShock, 1996
- Interleukin 10 (IL-10) upregulates IL-1 receptor antagonist production from lipopolysaccharide-stimulated human polymorphonuclear leukocytes by delaying mRNA degradation.The Journal of Experimental Medicine, 1994
- Neutrophil and Nonneutrophil-Mediated Injury in Intestinal Ischemia-ReperfusionAnnals of Surgery, 1993
- Interleukin-10Annual Review of Immunology, 1993
- Interleukin 10 protects mice from lethal endotoxemia.The Journal of Experimental Medicine, 1993
- Interleukin 10 reduces the release of tumor necrosis factor and prevents lethality in experimental endotoxemia.The Journal of Experimental Medicine, 1993
- Biological properties of interleukin 10Immunology Today, 1992
- The role of IL-10 in crossregulation of TH1 and TH2 responsesImmunology Today, 1991
- Evidence for Tumor Necrosis Factor-induced Pulmonary Microvascular Injury After Intestinal Ischemia—Reperfusion InjuryAnnals of Surgery, 1990
- Role of tumor necrosis factor-alpha in the pathophysiologic alterations after hepatic ischemia/reperfusion injury in the rat.Journal of Clinical Investigation, 1990