JunB Forms the Majority of the AP-1 Complex and Is a Target for Redox Regulation by Receptor Tyrosine Kinase and G Protein-coupled Receptor Agonists in Smooth Muscle Cells
Open Access
- 1 February 1999
- journal article
- Published by Elsevier
- Vol. 274 (9) , 6003-6010
- https://doi.org/10.1074/jbc.274.9.6003
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Protein Tyrosine Kinase Activity Is Required for Oxidant-Induced Extracellular Signal-Regulated Protein Kinase Activation and c-fos and c-jun ExpressionCellular Signalling, 1997
- Stimulated Mitogen-activated Protein Kinase Is Necessary but Not Sufficient for the Mitogenic Response to Angiotensin IIPublished by Elsevier ,1996
- Activation of Mitogen-activated Protein Kinase by H2O2Journal of Biological Chemistry, 1996
- Reactive Oxygen Intermediates Mediate Angiotensin II-induced c-Jun•c-Fos Heterodimer DNA Binding Activity and Proliferative Hypertrophic Responses in Myogenic CellsJournal of Biological Chemistry, 1995
- Production of hydrogen peroxide by transforming growth factor-beta 1 and its involvement in induction of egr-1 in mouse osteoblastic cells.The Journal of cell biology, 1994
- Arachidonic Acid Metabolites in bFGF-, PDGF-, and Serum-Stimulated Vascular Cell GrowthExperimental Cell Research, 1994
- The stress-activated protein kinase subfamily of c-Jun kinasesNature, 1994
- Redox Regulation of Fos and Jun DNA-Binding Activity in VitroScience, 1990
- Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factorCell, 1987
- Stimulation of murine lymphocyte responses by cationsCellular Immunology, 1986