Regulation by reactive oxygen species of interleukin-1β, nitric oxide and prostaglandin E2 production by human chondrocytes
- 1 July 2002
- journal article
- Published by Elsevier in Osteoarthritis and Cartilage
- Vol. 10 (7) , 547-555
- https://doi.org/10.1053/joca.2002.0789
Abstract
No abstract availableThis publication has 30 references indexed in Scilit:
- Nitric oxide reactivity and mechanisms involved in its biological effectsPharmacological Research, 2000
- N -Acetyl- l -Cysteine Potentiates Interleukin-1β Induction of Nitric Oxide SynthaseHypertension, 2000
- Inhibition by nitric oxide‐releasing compounds of prostacyclin production in human endothelial cellsBritish Journal of Pharmacology, 1998
- Redox Signals and NF-κB Activation in T CellsFree Radical Biology & Medicine, 1998
- Nitric oxide amplifies interleukin 1-induced cyclooxygenase-2 expression in rat mesangial cells.Journal of Clinical Investigation, 1996
- The increased synthesis of inducible nitric oxide inhibits IL-1ra synthesis by human articular chondrocytes: possible role in osteoarthritic cartilage degradationOsteoarthritis and Cartilage, 1996
- Inhibition of Ribonucleotide Reductase by Nitric Oxide Derived from Thionitrites: Reversible Modifications of Both SubunitsBiochemistry, 1995
- Influence of Antioxidants on Arachidonic Acid Metabolism and Platelet FunctionBiochemical Medicine and Metabolic Biology, 1994
- PRODUCTION OF ACTIVE OXYGEN SPECIES BY ISOLATED HUMAN CHONDROCYTESRheumatology, 1993
- Nitric oxide, an endothelial cell relaxation factor, inhibits neutrophil superoxide anion production via a direct action on the NADPH oxidase.Journal of Clinical Investigation, 1992