The early decay of long-term potentiation in the hippocampal CA1 region in vitro is reduced by activators of protein kinase C
- 1 June 1990
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 521 (1-2) , 355-358
- https://doi.org/10.1016/0006-8993(90)91567-z
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- NMDA receptors - their role in long-term potentiationPublished by Elsevier ,2003
- Onset Characteristics of Long‐Term Potentiation in the Guinea‐Pig Hippocampal CA1 Region in VitroEuropean Journal of Neuroscience, 1989
- Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiationNature, 1989
- Physiological mechanisms underlying long-term potentiationTrends in Neurosciences, 1988
- Synaptic memory moleculesNature, 1988
- NMDA application potentiates synaptic transmission in the hippocampusNature, 1988
- Phorbol ester-induced synaptic potentiation differs from long-term potentiation in the guinea pig hippocampus in vitroNeuroscience Letters, 1988
- Potentiation of synaptic transmission in the hippocampus by phorbol estersNature, 1986
- Translocation of Protein Kinase C Activity May Mediate Hippocampal Long-Term PotentiationScience, 1986
- Facilitation of hippocampal long‐lasting potentiation by GABA antagonistsActa Physiologica Scandinavica, 1985