K-ras Gene Mutation Enhances Motility of Immortalized Airway Cells and Lung Adenocarcinoma Cells via Akt Activation
- 1 January 2004
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 164 (1) , 91-100
- https://doi.org/10.1016/s0002-9440(10)63100-8
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Hepatocyte Growth Factor Inhibits Anoikis in Head and Neck Squamous Cell Carcinoma Cells by Activation of ERK and Akt Signaling Independent of NFκBJournal of Biological Chemistry, 2002
- Phosphorylation of Akt/PKB is required for suppression of cancer cell apoptosis and tumor progression in human colorectal carcinomaCancer, 2002
- Functional roles of Akt signaling in mouse skin tumorigenesisOncogene, 2002
- Akt and PTEN: New diagnostic markers of non‐small cell lung cancer?Journal of Cellular and Molecular Medicine, 2001
- Akt-Mediated Phosphorylation of the G Protein-Coupled Receptor EDG-1 Is Required for Endothelial Cell ChemotaxisMolecular Cell, 2001
- Genetic model of multi-step breast carcinogenesis involving the epithelium and stroma: clues to tumour-microenvironment interactionsHuman Molecular Genetics, 2001
- Substrate Specificity of αvβ3Integrin-mediated Cell Migration and Phosphatidylinositol 3-Kinase/AKT Pathway ActivationJournal of Biological Chemistry, 2000
- Ki-ras and p53 mutations are early and late events, respectively, in urethane-induced pulmonary carcinogenesis in A/J miceMolecular Carcinogenesis, 1996
- SV40-lnduced Immortalization of Human CellsCritical Reviews™ in Oncogenesis, 1994
- Mutational Activation of the K-rasOncogeneNew England Journal of Medicine, 1987