Pertussis-toxin-sensitive Gα subunits selectively bind to C-terminal domain of neuronal GIRK channels: evidence for a heterotrimeric G-protein-channel complex

This publication has 74 references indexed in Scilit:

• Gi protein activation in intact cells involves subunit rearrangement rather than dissociation
  Proceedings of the National Academy of Sciences, 2003
• Defective Potassium Channel Kir2.1 Trafficking Underlies Andersen-Tawil Syndrome
  Journal of Biological Chemistry, 2003
• The Dynamics of Formation and Action of the Ternary Complex Revealed in Living Cells Using a G-protein-gated K+ Channel as a Biosensor
  Published by Elsevier ,2003
• A glutamate residue at the C terminus regulates activity of inward rectifier K ⁺ channels: Implication for Andersen's syndrome
  Proceedings of the National Academy of Sciences, 2002
• The γ‐aminobutyric acid receptor B, but not the metabotropic glutamate receptor type‐1, associates with lipid rafts in the rat cerebellum
  Journal of Neurochemistry, 2001
• Disruption of cytoskeletal integrity impairs Gi-mediated signaling due to displacement of Gi proteins
  The Journal of cell biology, 2001
• Interaction Sites of the G Protein β Subunit with Brain G Protein-coupled Inward Rectifier K+ Channel
  Journal of Biological Chemistry, 2001
• On the role of endogenous G‐protein βγ subunits in N‐type Ca²⁺ current inhibition by neurotransmitters in rat sympathetic neurones
  The Journal of Physiology, 1998
• G protein .beta..gamma. subunits from bovine brain and retina: equivalent catalytic support of ADP-ribosylation of .alpha. subunits by pertussis toxin but differential interactions with Gs.alpha.
  Biochemistry, 1989
• Uncoupling of cardiac muscarinic and β-adrenergic receptors from ion channels by a guanine nucleotide analogue
  Nature, 1985