A Defect in the Kv Channel-Interacting Protein 2 (KChIP2) Gene Leads to a Complete Loss of Ito and Confers Susceptibility to Ventricular Tachycardia
Top Cited Papers
- 1 December 2001
- Vol. 107 (6) , 801-813
- https://doi.org/10.1016/s0092-8674(01)00588-8
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Conserved Kv4 N-terminal Domain Critical for Effects of Kv Channel-interacting Protein 2.2 on Channel Expression and GatingJournal of Biological Chemistry, 2001
- The Brugada syndrome: diagnostic criteria and cellular mechanismsEuropean Heart Journal, 2001
- Molecular basis of functional voltage‐gated K+ channel diversity in the mammalian myocardiumThe Journal of Physiology, 2000
- Modulation of A-type potassium channels by a family of calcium sensorsNature, 2000
- Signaling Pathways for Cardiac Hypertrophy and FailureNew England Journal of Medicine, 1999
- Expression of theMf1 gene in developing mouse hearts: Implication in the development of human congenital heart defectsDevelopmental Dynamics, 1999
- Cloning and Expression of a Novel K+ Channel Regulatory Protein, KChAPPublished by Elsevier ,1998
- Electrophysiological abnormalities and arrhythmias in alpha MHC mutant familial hypertrophic cardiomyopathy mice.Journal of Clinical Investigation, 1997
- βSubunits Promote K+ Channel Surface Expression through Effects Early in BiosynthesisNeuron, 1996
- Inactivation properties of voltage-gated K+ channels altered by presence of β-subunitNature, 1994